i.e. it inhibits the growth of tumour cells. So before imatinib can be used against biofilms in humans with aspergillosis, its effectiveness as an Agd3 inhibitor would have to be enhanced. "Otherwise

journals Genome Medicine and Kidney International that show the molecular pattern of acute damage to human kidney cells. Direct zoom into the cell "Acute kidney damage is associated with a wide variety of

in the blood of AIH patients. "They have the special property of being able to bind with very many human proteins," explains the gastroenterologist. New test closes diagnostic gap These so-called polyreactive

to evaluate data," says Dr Jack. However, medical decisions are not made by the system, but by the human being, the physician emphasises. "ELISE is just a kind of co-pilot that, as a digital companion, frees

sms of hypertrophic cardiomyopathy, physiology and pathophysiology of skeletal muscle fibers and human cardiomyocytes, molecular aspects of contraction and motility

Hereditary Chronic Pancreatitis and Their Families: A Qualitative Triangulation. European Journal of Human Genetics 29(1):29-38. Seidlein AH*, Hannich A*, Nowak A, Gründling M, Salloch S (2020): Ethical aspects

single-cell analysis, and on the other hand, the analysis of complex metagenomes based on different human tissues and body fluids using optimized laboratory processes and an analysis pipeline developed in-house

huge impact for both the optimization of differentiation protocols as well as for drug-screening are human iPS reporter cell lines which are generated using TALEN-based homologues recombination ( RG Martin

Shaheed IB, Mosimann C, Diecke S, Schmidt-Ott KM . The centrosomal protein 83 (CEP83) regulates human pluripotent stem cell differentiation toward the kidney lineage. Elife, 11:e80165, 2022. Hinze C, [...] Rajewsky N, Schmidt-Ott KM . Single-cell transcriptomics reveals common epithelial response patterns in human acute kidney injury. Genome Medicine, 14:103, 2022. Hinze C, Karaiskos N, Boltengagen A, Walentin

levels have been shown to decrease as the disease progresses. "In studies on the mouse model and in human liver cells, we have already been able to show that the therapeutic mRNA can be used to restore HFN4α