and/or indirect microglia/neuron-interactions and subsequently controls neuronal excitability and gene expression. We aim at revealing the therapeutic potential of microglia depletion on inflammation-related
and/or indirect microglia/neuron-interactions and subsequently controls neuronal excitability and gene expression. We aim at revealing the therapeutic potential of microglia depletion on inflammation-related
to answer whether early traumatic childhood experiences and the methylation of exon IV of the BDNF gene have an influence on the differential effectiveness of the two treatments. Mediator analyzes are used
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Holger Prokisch, Tim Ripperger, and David Malkin. 2022. “ Heterozygous BRCA1/2 and Mismatch Repair Gene Pathogenic Variants in Children and Adolescents with Cancer .” JNCI: Journal of the National Cancer [...] Nilesh J. Samani. 2009. “Genome-Wide Haplotype Association Study Identifies the SLC22A3-LPAL2-LPA Gene Cluster as a Risk Locus for Coronary Artery Disease.” Nature Genetics 41(3):283–85. doi: 10.1038/ng [...] Novel Genetic Variant Predisposing to Coronary Artery Disease in the Region of the PSRC1 and CELSR2 Genes on Chromosome 1 Associates with Serum Cholesterol.” Journal of Molecular Medicine 86(11):1233–41.
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