Nugraha B, Anwar SL, Gutenbrunner C, Korallus C. Polymorphism of brain-derived neurotrophic factor genes is associated with anxiety and body mass index in fibromyalgia syndrome patients. BMC Res Note 2020;
Bange FC, Wehling C, Kirschfink M, Bergmann C, Pape L. Systemic complement activation and complement gene analysis in enterohaemorrhagic Escherichia coli -associated paediatric haemolytic uraemic syndrome
exon 12, a heterozygous in-frame deletion in exon 3 and a missense mutation in exon 11 of the BMPR2 gene. Stem Cell Res. 2021;55:102488. https://pubmed.ncbi.nlm.nih.gov/34388490/ . Kokeny G, Calvier L, Hansmann
Arbeiten (Bachelor- und Masterarbeiten, Laborpraktika): Screening von relevanten Virusgenomen auf Gene, die für PDE-/Nukleasehomologe kodieren und Produktion/Reinigung der entsprechenden Enzyme Identifizierung
Gottlieb J, Welte T, Braubach P, Laenger F, Hoeper MM, Kuehnel MP, Jonigk DD. Comparative Analysis of Gene Expression in Fibroblastic Foci in Patients with Idiopathic Pulmonary Fibrosis and Pulmonary Sarcoidosis
P. & Hunt, D.M. (1995) Cloning vectors for the production of proteins in Dictyostelium discoideum Gene 162 , 129-134. (Abstract) (pdf, 436 kB) Schröder, R.R., Manstein, D.J., Jahn, W., Holden, H.M., Rayment [...] Biol. 137 , 359-367. (Abstract) Manstein, D.J., Titus, M.A., De Lozanne, A. & Spudich, J.A. (1989) Gene replacement in Dictyostelium: generation of myosin null mutants EMBO J. 8 , 923-932. (Abstract) Manstein
Daniel C, Kispert A, Haffner D, Weber RG: Mutations in the leukemia inhibitory factor receptor (LIFR) gene and Lifr deficiency cause urinary tract malformations. Hum Mol Genet 2017; 26(9): 1716-1731 Kröger [...] Schuster-Gossler K, Kühnel MP , Hegermann J, Ueffing M, Gossler A: 1700012B09Rik, a FOXJ1 effector gene active in ciliated tissues of the mouse but not essential for motile ciliogenesis. Dev Biol 2017;
and/or indirect microglia/neuron-interactions and subsequently controls neuronal excitability and gene expression. We aim at revealing the therapeutic potential of microglia depletion on inflammation-related
and/or indirect microglia/neuron-interactions and subsequently controls neuronal excitability and gene expression. We aim at revealing the therapeutic potential of microglia depletion on inflammation-related
to answer whether early traumatic childhood experiences and the methylation of exon IV of the BDNF gene have an influence on the differential effectiveness of the two treatments. Mediator analyzes are used